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. 2021 Jul 1;12:656115. doi: 10.3389/fphar.2021.656115

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Copyright © 2021 Wang, Tang, Zhao, Shen, Lin, Zhu and Han.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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SNS inhibits pathological microvessel and angiogenesis-associated signaling pathways in liver tissue of mice with liver fibrosis. (A) Image analysis of fluorescence intensity of CD 34 (pathological microvessels) stained with IF in hepatic tissue of mice with CCl₄-induced liver fibrosis. (B) Effect of SNS on VEGF expression in liver tissue. (C) Effects of SNS on VEGFR1 and VEGFR2 protein levels in liver tissue. (D) Effects of SNS on Flt1 and Kdr mRNA levels in liver tissue. Data are presented as mean ± SD (n = 6). *p < 0.05, **p < 0.01, ***p < 0.001 compared to control or model group.