Table 3.
Correlation between resistance phenotype and genotype among studied LAB species (n = 40).
Strains | Resistance phenotypea | Resistance genotype |
---|---|---|
L. fermentum KKP 2020 | TE | n.d. |
L. fermentum KKP 830 | n.d. | |
L. brevis Pat1 | n.d. | |
L. brevis Solaris | n.d. | |
L. brevis Pap3/4 | n.d. | |
L. farraginis E/J | n.d. | |
P. pentosaceus Pom7 | n.d. | |
L. buchneri KKP 2047p | K | aph(3″)-IIIa |
L. fermentum Sieger | n.d. | |
L. plantarum KKP 815 | n.d. | |
L. plantarum KKP 835 | aph(3″)-IIIa | |
L. plantarum KKP 870 | aph(3″)-IIIa | |
L. plantarum KKP 872 | n.d. | |
L. plantarum KKP 2021p | n.d. | |
L. plantarum KKP 1821 | n.d. | |
L. plantarum KKP 1822 | aph(3″)-IIIa | |
L. plantarum ATCC 8287 | n.d. | |
L. delbrueckii PCM 490 | n.d. | |
P. pentosaceus MA | K – TE | n.d. |
L. salivarius KKP 1828 | K – S | n.d. |
L. rhamnosus KKP 849 | K – CH | n.d. |
L. plantarum KKP 804 | A | n.d. |
L. rhamnosus B/J | GM – K | n.d. |
L. acidophilus PCM 2499 | TE – S | tet(K), str(A)/str(B) |
P. pentosaceus WN1 | n.d. | |
E. durans KKP 1586 | E – CL | erm(B) |
E. faecium TR2 | TE – E | tet(M), msr(C), lnu(A) |
L. diolivorans KKP 2036p | TE – K – S | aph(3″)-IIIa |
L. salivarius KKP 1835 | tet(M), str(A)/str(B) | |
P. pentosaceus KapA | n.d. | |
P. pentosaceus AG | n.d. | |
P. acidilactici KKP 1839 | n.d. | |
L. johnsonii KKP 878 | TE – CH – S | n.d. |
L. agilis KKP 1834 | GM – K – S – E | aph(3″)-IIIa, msr(C) |
L. fermentum KKP 811, KKP 830, KKP 843 | K | aph(3″)-IIIa |
L. plantarum KKP 870, KKP 872 | CL | lnu(A) |
L. plantarum KKP 2021p | CL | erm(B) |
GM, gentamicin; K, kanamycin; TE, tetracycline; CH, chloramphenicol; A, ampicillin; E, erythromycin; CL, clindamycin; S, streptomycin; n.d., tested resistance genes not detected. The strains carrying a resistance gene but phenotypically resistant only in line to cut-off values adopted in previous EFSA guideline (2012) are in bold.