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. 2021 Jul 6;9(19):4998–5006. doi: 10.12998/wjcc.v9.i19.4998

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©The Author(s) 2021. Published by Baishideng Publishing Group Inc. All rights reserved.

This article is an open-access article that was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution NonCommercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial. See: http://creativecommons.org/Licenses/by-nc/4.0/

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Involvement of epithelial-mesenchymal transition in therapy resistance in pancreatic ductal adenocarcinoma. Epithelial-mesenchymal transition (EMT) is induced by various factors including signaling pathways, EMT-activating transcription factors (EMT-TFs), microRNAs, or microenvironment. Promotion of the EMT program enhances the chemoresistance in pancreatic ductal adenocarcinoma. EMT: Epithelial-mesenchymal transition; PDAC: Pancreatic ductal adenocarcinoma; EMT-TFs: Epithelial-mesenchymal transition-activating transcription factors; CAFs: Cancer-associated fibroblasts; PSCs: Pancreatic stellate cells.