Table 1. Summary of findings in structural imaging.

HV: healthy variants; AD: Alzheimer's disease; DLB: Dementia with Lewy bodies; PDD: Parkinson's disease with dementia; MRI: magnetic resonance imaging

Author	Study performed	Subjects	Findings
Barber et al.	T(1)-weighted, T(2)-weighted, and proton density MRI	25 AD and 27 DLB	AD is characterized by more pronounced atrophy in the temporal lobe, amygdala, and hippocampus, which is relatively spared in DLB. Periventricular hyperintensities correlate well with age while deep white matter hyperintensities correlate well with a history of hypertension [9].
Ballmaier et al.	MRI	29 AD, 16 DLB, 38 HV	More pronounced grey matter atrophy in orbitofrontal and temporal regions in AD compared to that of DLB [10].
Burton et al.	Voxel-based morphometry	26 PDD, 31 PD, 28 AD, 17 DLB, 36 HV	Extensive volume loss in PDD involving the frontal lobe, temporal lobe, including the hippocampal and parahippocampal gyrus, occipital lobe, while PD with dementia showed such changes only confined to the frontal lobe. No significant differences could be observed between PDD and DLB [11].
Sanchez- Castaneda et al.	Voxel-based morphometry	12 DLB and 15 PDD	Visual symptoms are increasingly found to be associated with a higher amount of grey matter loss. In DLB, this is found to be focused around the right inferior frontal gyrus while in PDD it is more pronounced around the region of the left orbitofrontal lobe [12].
Lebedev et al.	Sparse partial least squares (SPLS) classification of cortical thickness measurements in MRI	Two cohorts involving 97 AD and DLB subjects	Subjects with AD showed atrophy in the mid-anterior temporal, occipital, and subgenual cingulate cortex while DLB was characterized by a unique pattern of cortical thinning in the regions of dorsal cingulate, posterior temporal, and lateral orbitofrontal area [13].