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. 2020 Jul 30;15(3):1403–1411. doi: 10.1007/s11682-020-00339-x

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — FOXP2 expression in the prefrontal cortex of male donor subjects with schizophrenia versus controls (A); accounting for the FOXP2 rs2396753 polymorphism in the total samples (B); taking into consideration the interaction between this polymorphism and the clinical groups (C). FOXP2 expression is indicated as fold changes relative to the control group (A), AA genotype (B) and AA genotype of the control group (C) considering its expression as 1 in each case. Asterisk indicates the degree of significance (* p < 0.05) using an unpaired t test (A) and one-way ANOVA with Bonferroni multiple comparison tests (B and C). Error bars represent standard deviation