Table 1.

Characteristics of Severe Acute Pancreatitis-Associated Cardiac Injury

Characteristics	Supplements	Check Type	References
Cardiomyocytes: a rough type, hypoxia, edema, hypertrophy and apoptosis	Over contracted myofibrils caused by supercharging with calcium Deficiencies in the sarcolemma structure. Affection of the microcirculation vessels structure	Histopathological changes	[11,30,38,45,47,67,82,88,116]
Myocardium stroma: edema and collagenization	Deficiencies in the sarcoma structure compensatory response of connective tissue to lesions		[11,39,46,47,82]
Electrocardiogram	Arrhythmia: prolonged QTc interval	Laboratory and imaging changes	[5,135,155]
Biochemical examination	CK-MB, LDH, cTnI		[114,161]
Echocardiography	Myocardial contractile dysfunction: decreased ejection fraction, poor contraction response to volume load, decreased peak systolic pressure/end-systolic volume ratio, enlarged heart and pericardial effusion		[4,30]
Other complications	Increased blood flow and permeability of pulmonary capillaries, increased adhesion and infiltration of leukocytes and thickening of alveolar septum	Histopathological changes	[14,52,106,119,122,165]
	Decreased renal blood flow and acute renal failure		[6,122,164]