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. 2021 Jul 15;15:17534666211028072. doi: 10.1177/17534666211028072

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© The Author(s), 2021

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (https://creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 5. — miR-18a-5p reversed the influence of CASC2 on cell apoptosis and inflammatory in 16HBE cells. (a) miR-18a-5p mimic transfection increased the level of miR-18a-5p remarkably. (b) Influence of miR-18a-5p on cell viability. (c) Effect of miR-18a-5p on cell apoptosis. (d) The levels of IL-6, IL-8, IL-1β, and TNF-α in cells after miR-18a-5p upregulated.

***p < 0.001, compared with CSE+miR-NC group.

^##p < 0.01, ^###p < 0.001, compared with CSE+pcDNA group.

^{&p < 0.05, &&}p < 0.01, ^&&&p < 0.001, compared with CSE+CASC2 group.

Differences between groups were compared using one-way analysis of variance.

CASC2, cancer susceptibility candidate 2; CSE, cigarette smoke extract; NC, negative control; pcDNA, plasmid cloning DNA.