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. 2020 Jun 17;11(1):661–678. doi: 10.1080/21655979.2020.1771068

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© 2020 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 1. — Genes were up-regulated in tumor-infiltrated dendritic cells and enriched in pathways associated with pain. The dendritic cells were isolated from tumor or juxtatumor tissues, and RNA was extracted from dendritic cells and used to RNA-seq. (a) The transcriptome of tumor-infiltrated CD1c⁻ dendritic cells. (b) The transcriptome of tumor-infiltrated CD1c⁺ dendritic cells. (c) The enrichment of genes from 1.5-fold up-regulated genes of tumor-infiltrated CD1c⁻ dendritic cells in pain associated pathways. (d) The enrichment of genes from 1.5-fold up-regulated genes of tumor-infiltrated CD1c⁺ dendritic cells in pain associated pathways. Enrichment index was calculated as ratio of actually up-regulated genes to expectedly up-regulated genes. A.U. was arbitrary unit.