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. 2021 Jul 7;12:720734. doi: 10.3389/fendo.2021.720734

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Copyright © 2021 Dao and François

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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CB1R inhibition could be a new therapeutic target in chronic kidney diseases and renal fibrosis, in both metabolic and non-metabolic nephropathies. Both in vitro and in vivo experiments have been taken into account. ACR, urinary albumin-to-creatinine ratio; CB1R, cannabinoid receptor type 1; CKD, chronic kidney disease; IF/TA, interstitial fibrosis and tubular atrophy; iNOS, inducible nitric oxide synthase; pCB1Rko, podocyte-specific deletion of CB1R; RPTC, renal proximal tubular cells.