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. 2021 Jul 21;12(8):722. doi: 10.1038/s41419-021-04013-y

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — A–B PPM1G was highly expressed in the HCC samples as compared with adjacent normal tissues. The IHC experiments were performed in HCC samples and adjacent normal tissues using antibodies against PPM1G (A). The statistical comparison of the PPM1G levels between HCC samples and adjacent normal tissues was shown (B). C The mRNA levels of PPM1G were significantly higher in HCC tissues than adjacent normal tissues or normal liver tissues in the GEO database. D The levels of PPM1G were increased in the advanced stages of HCC. The PPM1G scores in the different stages of HCC were shown. E The mRNA levels of PPM1G were highly expressed in HCC. The levels of PPM1G in the TCGA-LIHC cohort were analyzed.