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. 2021 Jul 16;27:78. doi: 10.1186/s10020-021-00338-8

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — EVs-miR-182-5p exerts promoting effects on HUVECs proliferation, migration and angiogenesis. HUVECs were treated with EVs + miR-182-5p inhibitor. A miR-182-5p expression of HUVECs detected by RT-qPCR, the statistical power was 1; B VEGF protein expression of HUVECs detected by Western blot analysis, the statistical power was 1; C The proliferation of HUVECs detected by CCK-8 assay, the statistical power was 1; D Migration ability of HUVECs detected by scratch test, the statistical power was 1; E Migration ability of HUVECs detected by Transwell assay, the statistical power was 1; F Vessel-like tube formation in vitro of HUVEC, the statistical power was 1. *p < 0.05, ***p < 0.001, ****p < 0.0001 compared with HUVEC; #p < 0.05, ##p < 0.01, ###p < 0.001 compared with HUVEC treated with EVs + inhibitor NC. The experiment was conducted three times independently