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. 2021 May 6;40(29):4746–4758. doi: 10.1038/s41388-021-01800-x

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — NB4 cells infected with a control lentivirus (NB4control), a MYBΔ3-encoding lentivirus (NB4MYBΔ3) or a C/EBPβ-encoding lentivirus (NB4C/EBPβ) were treated for 72 h with the indicated concentrations of AIB or HA. The cells were then stained with antibodies against CD11b (A) or with annexin V and propidium iodide (B) and analyzed by flow cytometry. Bars (A) indicate the percentage of CD11b positive cells (with standard deviations) of the viable cells. Asterisks indicate statistical significance (**p < 0.01, ***p < 0.001, Student’s t test). Bar charts (B) indicate the percentages (with standard deviations) of living (A-/PI-), early (A + /PI-) and late (A + /PI + ) apoptotic and necrotic (A-/PI + ) cells. The chemical structures of AIB and HA are shown in (A).