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. 2021 Mar 28;20(4):1337–1351. doi: 10.1007/s10237-021-01447-3

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — a, b Pulsewave Doppler measurement of velocity at the proximal outflow tract with standard deviation demarcated in grey, for a HH25, control (n = 6) and LAL hearts (n = 5) and b HH28, control (n = 5) and LAL hearts (n = 5). Peak velocity for HH25 was significantly different between control and LAL (p < 0.01) c outflow tract for control and LAL (P < 0.05). d Cardiac output of the HH25 embryonic hearts