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. 2021 Jul 22;12(8):726. doi: 10.1038/s41419-021-04015-w

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© The Author(s) 2021, corrected publication 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 8 — A Hsa_circ_0001747 was downregulated in EBSS treatment. B Impediment of autophagy displayed no effect on the expression of hsa_circ_0001747. C Western blotting confirmed knockdown of hsa_circ_0001747 enhanced autophagy-related protein expression. D Knockdown of hsa_circ_0001747 facilitated the autophagy flux, including the autophagosomes (yellow dots) and autophagolysosomes (red dots) formation in 22Rv1 cells. E Knockdown of hsa_circ_0001747 promoted the formation of autophagosomes in 22Rv1 cells. F CQ treatment rescued the promotion of proliferation induced by knockdown of hsa_circ_0001747 in PCa. Error bar indicates mean ± SD. “ns” and “#” indicates not significant. *P < 0.05, **P < 0.01, ***P < 0.001.