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. 2021 Jul 12;46(3):187. doi: 10.3892/or.2021.8138

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Copyright: © Jin et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — 786-O-EVs are the communication mechanism of MALAT1 in RCC cells. (A) The expression of candidate lncRNAs in the EVs evaluated through the exoRBase database (http://www.exorbase.org/); the abscissa represents the EVs from different tumors, and the ordinate represents the expression of different lncRNAs; the left side shows the diagram. NP, normal person; CHD, coronary heart disease; CRC, colorectal cancer; HCC, hepatocellular carcinoma; PAAD, pancreatic adenocarcinoma; WhB, whole blood. (B) Expression of MALAT1 in ACHN and A498 cells treated with 786-O-EVs was detected using qPCR. (C) Expression of MALAT1 in 786-O, ACHN and A498 cells after treatment of RNase and Triton X-100 was detected using qPCR. Each experiment was repeated three times independently. Data in panels B and C were analyzed using independent t-test or one-way ANOVA, followed by Tukey's multiple comparisons test. **P<0.01. EVs, extracellular vesicles; MALAT1, metastasis-associated lung adenocarcinoma transcript 1.