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. 2021 Jul 12;22(3):658. doi: 10.3892/ol.2021.12919

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Copyright: © Themistocleous et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 5. — Expression levels of HKA in cancer compared with in healthy tissues across 24 types of cancer. Downregulation of HKA expression was markedly observed in esophageal carcinoma and stomach adenocarcinoma, and was less distinct in thyroid carcinoma. Overall expression in all other tissues appeared to be limited. Data were collected and permitted for publication from the UALCAN resource. TPM, transcripts per million; TCGA, The Cancer Genome Atlas; HKA, H⁺/K⁺ ATPase; BLCA, bladder urothelial carcinoma; BRCA, breast invasive carcinoma; CESC, cervical squamous cell carcinoma; CHOL, cholangiocarcinoma; COAD, colon adenocarcinoma; ESCA, esophageal carcinoma; GBM, glioblastoma multiforme; HNSC, head and neck squamous carcinoma; KICH, kidney chromophobe; KIRC, kidney renal clear cell carcinoma; KIRP, kidney renal papillary cell carcinoma; LIHC, liver hepatocellular carcinoma; LUAD, lung adenocarcinoma; LUSC, lung squamous cell carcinoma; PAAD, pancreatic adenocarcinoma; PRAD, prostate adenocarcinoma; PCPG, pheochromocytoma and paragangliosarcoma; READ, rectal adenocarcinoma; SARC, sarcoma; SKCM, skin cutaneous melanoma; THCA, thyroid carcinoma; THYM, thymoma; STAD, stomach adenocarcinoma; UCEC, uterine corpus endometrial carcinoma.