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Telomere length dynamics in germinal, stem, somatic, and cancer cells according to the presence of telomerase activity. In germinal and stem cells, active telomerase can offset telomere attrition due to cell divisions. In somatic cells, telomerase is inactive, and telomere length is shortened until it reaches a threshold that triggers replicative senescence. Cancer cells can bypass cell senescence by the reactivation of telomerase.
