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. 2021 Jul 23;11:15073. doi: 10.1038/s41598-021-94499-5

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Expression of ERRα in the radio-resistant compartment mediates resistance to DSS colitis. (a) Colon length of bone marrow chimera mice on day 8 following DSS treatment. Data represent the mean ± SEM of n = 8–10 mice. Statistical analysis was performed with Student’s t-test, *p < 0.05, **p < 0.01. ns, not significant. (b) Representative photographs of colon and cecum on day 8 following DSS treatment. (c) Representative Hematoxylin and eosin (H&E) staining of colon sections derived from bone marrow chimera mice on day 8 following DSS treatment. (d) Intestinal tissue damage and erosion of bone marrow chimera mice on day 8 following DSS treatment, quantified by measuring the length of healthy, damaged, eroded sections, and expressed as a percentage of colon length. Data represent the mean ± SEM of 8–10 mice/genotype. Statistical analysis was performed using two-way analysis of variance (ANOVA, *p < 0.05, **p < 0.01, ***p < 0.001. ns, not significant).