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. 2021 Jul 9;22(14):7372. doi: 10.3390/ijms22147372

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© 2021 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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Involvement of Helicobacter pylori in gastric lymphogenesis. The complex relationship between pathogen-related events, host factors and immune mechanisms plays a key role in the development of gastric lymphoma. APRIL: a proliferation-inducing ligand; TNF alpha: Tumor Necrosis Factor α; GSTT1: glutathione s-transferases theta 1, CTLA4: Cytotoxic T-lymphocyte-associated protein 4; IL-22: interleukin-22; IL-18: interleukin-18; IL-8: interleukin-8; IL-6: interleukin-6; PD-L1: programmed death-ligand 1; PD-1: Programmed cell death protein 1; MALT: mucosa-associated lymphoid tissue lymphoma; DLBCL: diffuse large B-cell lymphoma.