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. 2021 Jul 6;97(1):e1–e12. doi: 10.1212/WNL.0000000000012175

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Copyright © 2021 The Author(s). Published by Wolters Kluwer Health, Inc. on behalf of the American Academy of Neurology.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivatives License 4.0 (CC BY-NC-ND), which permits downloading and sharing the work provided it is properly cited. The work cannot be changed in any way or used commercially without permission from the journal.

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Scatterplots of antibody index (AI) after the exclusion of patients with relapse prevention therapy or with CSF-restricted anti-myelin oligodendrocyte glycoprotein antibody (MOG-IgG) are shown. Both the distribution of AI value and the prevalence of AI value ≥1.5 or >4 were still significantly higher in the MOG-IgG seropositive patients than in the anti–aquaporin-4 antibodies (AQP4-IgG) seropositive patients. Diagonal solid lines represent the AI values of 1.5 and 4.0. IgG = immunoglobulin G; = quotient for each specific antibody (i.e., , ); = upper line of the distribution of patients without intrathecal IgG synthesis on the Reibergram.

Inline graphic — Scatterplots of antibody index (AI) after the exclusion of patients with relapse prevention therapy or with CSF-restricted anti-myelin oligodendrocyte glycoprotein antibody (MOG-IgG) are shown. Both the distribution of AI value and the prevalence of AI value ≥1.5 or >4 were still significantly higher in the MOG-IgG seropositive patients than in the anti–aquaporin-4 antibodies (AQP4-IgG) seropositive patients. Diagonal solid lines represent the AI values of 1.5 and 4.0. IgG = immunoglobulin G; = quotient for each specific antibody (i.e., , ); = upper line of the distribution of patients without intrathecal IgG synthesis on the Reibergram.