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. 2021 Jul 14;8:679518. doi: 10.3389/fmed.2021.679518

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Copyright © 2021 Li, Veron and Tufro.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Glomerular Myo9A is S-nitrosylated in diabetic mice. (A) Proximity link assay IF signal (red) identifies abundant S-nitrosylated Myo9A (SNO-Myo9a) in glomeruli from uninduced diabetic mice with mild DKD (DM-iVEGF₁₆₄ - dox), whereas SNO-Myo9A is clearly reduced in glomeruli from induced diabetic mice with advanced DKD (DM-iVEGF₁₆₄, + dox). Dapi (blue) identifies cell nuclei. Scale bars = 50 μm. (B) Quantification of PLA IF signals, mean ± SD, n = 29–31/experimental group, unpaired t-test with Welch's correction, ****p < 0.0001.