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. 2021 Jul 3;12(17):5296–5309. doi: 10.7150/jca.56138

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miR-133-dependent facilitation of LPS-induced epithelial-mesenchymal transition. (A and B) The levels of molecular markers characteristic of EMT are indicated by western blot analyses upon an exposure of two independent oral cancer cells, OSC20 (left) and SAS (right) to LPS, in the presence (miR-133) and absence of stable expression of miR-133 (control). (C) The artificially generated gap regions of cultured SAS cell clusters with (miR-133) and without stable expression of miR-133 (control) are shown before (0H) and after 24 hour-long treatments of LPS (24H). The initial gap generated by scrapping is indicated with red lines in each image. Scale bar, 10 µm. (D) A quantitative measurement of the wound area is shown for each group before and after a 24 hour-long treatment of LPS. Mean ±SEM values are indicated for six different regions in cultures. One-way ANOVA test is performed. ***, P<0.001 for comparisons among the experimental conditions indicated.