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. 2021 Jul 30;4:928. doi: 10.1038/s42003-021-02427-0

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — a In the murine peritonitis model, mice were pre-treated with acetate (Ac) or PBS 30 min prior to intra-peritoneal S. aureus injection. b Acetate (Ac) treatment resulted in enhanced CD45⁺Ly6G⁺ neutrophil migration into the peritoneum compared to PBS-treated wild-type mice (black symbols) 6 h after infection. GPR43^−/− mice (blue symbols) showed reduced overall neutrophil migration into the peritoneum with no beneficial effect of acetate treatment. c Acetate-treated wild-type mice showed slightly reduced bacterial loads in the liver, whereas acetate treatment of GPR43^−/− mice did not influence bacterial loads. Data in panels b and c represent geometric means from six animals. *P < 0.05 significant difference versus the indicated PBS or wild-type control as calculated by Mann–Whitney U test.