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. 2021 Jul 31;21:724. doi: 10.1186/s12879-021-06425-9

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Sagittal T1 Fluid-attenuated inversion recovery (A), Axial T2 (B), and Coronal T2 images (C) were taken 4 weeks after the initial presentation: showing a 6cmx5cmx6cm multicystic mass lesion in the left temporal lobe with surrounding oedema. It is compressing the lateral ventricles, causing early hydrocephalus and midline shift to the right. Axial T2 were taken 4 weeks after the initial presentation (D), and 14 days after retreatment with antifungals and steroids (E): The multicystic mass in the left temporal lobe had increased in size (B) with more surrounding oedema, progressing obstructive hydrocephalus (B; red arrows), increased midline shift, effacement of basilar cisterns and compression of the brainstem