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. 2021 Jul 31;14:17562864211035543. doi: 10.1177/17562864211035543

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© The Author(s), 2021

This article is distributed under the terms of the Creative Commons Attribution 4.0 License (https://creativecommons.org/licenses/by/4.0/) which permits any use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 2. — Follow-up (within 2 months) axial T2-FLAIR (upper row) and axial contrast enhanced T1-SE (lower row) in patient 2, extensive progression of posterior accentuated, mostly subcortical PML-lesions is observed (upper row). Approximately 3 months after onset of the neurological symptoms, punctuate contrast enhancement (arrows) appear at the borders of the PML lesions suggesting the development of IRIS, corresponding to further clinical deterioration (lower row).

IRIS, immune reconstitution inflammatory syndrome; PML, progressive multifocal leukoencephalopathy.