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. Author manuscript; available in PMC: 2021 Aug 2.
Published in final edited form as: Cell Rep. 2021 Jun 22;35(12):109269. doi: 10.1016/j.celrep.2021.109269

Figure 7. FEZF2-EnR represses the increased expression of subcerebral neuronal genes in the corticothalamic neurons in the Tle4LacZ/LacZ mice.

Figure 7.

(A) Immunostaining of TLE4 or B-GAL, BCL11B, and BHLHB5 in the cortices of P7 Tle4+/+, Tle4LacZ/LacZ, and Tle4LacZ/LacZ; Fezf2-EnR mice. Scale bar: 100 μm.

(B) Quantifications of TLE4+ cells in the Tle4+/+ cortices and the B-GAL+ cells in Tle4LacZ/LacZ and Tle4LacZ/LacZ; Fezf2-EnR cortices.

(C) Quantifications of the numbers of BCL11B+BHLHB5+ cells.

(D) Immunostaining of TLE4, B-GAL, BCL11B, and FEZF2 in the cortices of P7 Tle4+/+, Tle4LacZ/LacZ, and Tle4LacZ/LacZ; Fezf2-EnR mice. Scale bar: 100 μm.

(E) Quantifications of the FEZF2+ cells by bin.

(F) Quantifications of the numbers of FEZF2+BCL11B+ cells. n = 3 brains per genotype, 3 sections per brain.

In all graphs, error bars represent ± SEM. Statistical significance was determined using one-way ANOVA followed by post hoc Tukey’s t test (*p < 0.05; **p < 0.01; ***p < 0.001). See also Figure S6.