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. 2021 Aug 2;12:4663. doi: 10.1038/s41467-021-25010-x

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — a Computed tomograghy angiography (CTA) showed the proximal M1 segment occlusion of the left middle cerebral artery (MCA) (black arrow) and the occlusion of the right internal carotid artery terminus (yellow arrow); b CT perfusion (CTP) showed bilateral infarct core (cerebral blood volume—CBV—map in c) and hypoperfusion (mean transit time—MTT—map in b), without treatable penumbra; d 24 h after, brain CT revealed bilateral malignant MCA infarcts; e portal vein thrombosis with extension to the left intrahepatic branches (black arrow); f subsegmental pulmonary artery thrombosis (white arrow).