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. 2021 Mar 19;28(8):2450–2464. doi: 10.1038/s41418-021-00762-7

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — a SNORD50A/B deletion frequency in 6 types of human cancers. b The association of SNORD50A/B deletion with poor patient survival in breast cancers (number of deletion group:188; number of wild-type group: 309), hepatocellular carcinomas (number of deletion group:28; number of wild-type group: 106) and lung adenocarcinomas (number of deletion group:21; number of wild-type group: 91). c The relationship between SNORD50A/B deletion and patient survival in breast cancers with mutant p53 (number of deletion group:41; number of wild-type group: 129) or wild-type p53 (number of deletion group:147; number of wild-type group: 180). d The relationship between p53 mutations and patient survival in breast cancers with deleted SNORD50A/B (number of mutation group:41; number of wild-type group: 147) or wild-type SNORD50A/B (number of mutation group:129; number of wild-type group: 180). All the data were from TCGA database.