Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2021 Aug 3;12(8):764. doi: 10.1038/s41419-021-04063-2

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

PMC Copyright notice

Fig. 6 — A The relative expression of ANLN-201 and ANLN-210 was examined in cells and exosomes by qRT-PCR. *P < 0.05, **P < 0.01. B The expression of ANLN-201 at mRNA level was examined in SCC-9 cells and exosomes when SCC-9 cells were transfected with si-HNRNPC. C The expression of ANLN-210 at mRNA level was examined in SCC-9 cells and exosomes when cells were transfected with si-HNRNPC and/or ANLN-210 overexpression. *P < 0.05, **P < 0.01. D The representative images of exosomes. Exosomes were extracted from SCC-9 cells transfected with ANLN-210 (210 O.E.) or exosomes were electrotransfected with ANLN-210 (210 E.T.). E Exosomal markers HSP70, CD63, and TSG101 were immunblotted in isolated exosomes. Calnexin which was expressing in cell lysates was used as the control. F The relative expression of ANLN-210 was examined in control, 210 O.E Exo. and 210 E.T. Exo groups. G Cell proliferation was performed in SCC-9 cell and HepG2 cells with the following treatment. PBS, exosomes derived from ANLN-sgRNA cells (Control Exo), exosomes derived from cells transfected with ANLN-210 (210 O.E. Exo), and exosomes electrotransfected with ANLN-210 (210 E.T. Exo). H Cell migration was analyzed in SCC-9 cells treated with the following treatment (PBS, Control Exo, 210 O.E. Exo, and 210 E.T. Exo).