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. 2021 May 21;14(8):767–778. doi: 10.1158/1940-6207.CAPR-20-0607

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©2021 The Authors; Published by the American Association for Cancer Research

This open access article is distributed under the Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International (CC BY-NC-ND 4.0) license.

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Figure 6. PD-1 blockage promoted recruitment of CD11c+ DCs into oral lesions of wild-type and mutant p53 mice. Representative IHC images of CD11c DCs in oral lesions. A significant difference of CD11+ cells was detected between untreated and PD1-treated mice groups. Immunostaining signal was defined as the number of positive cells per mm2. Right graphs: **, P <0.01; ***, P < 0.001; ****, P < 0.0001. Scale bar, 50 μm. — PD-1 blockage promoted recruitment of CD11c⁺ DCs into oral lesions of wild-type and mutant p53 mice. Representative IHC images of CD11c DCs in oral lesions. A significant difference of CD11⁺ cells was detected between untreated and PD1-treated mice groups. Immunostaining signal was defined as the number of positive cells per mm². Right graphs: **, P <0.01; ***, P < 0.001; ****, P < 0.0001. Scale bar, 50 μm.