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. 2021 Jun 1;12(3):813–838. doi: 10.1016/j.jcmgh.2021.05.017

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© 2021 The Authors

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

PMC Copyright notice

α-smooth muscle actin (α-SMA) expression in thioacetamide (TAA)-injured liver. Vehicle or thioacetamide (TAA)-administered mice were treated with lentivirus-delivered scramble or PRR shRNA (n = 6−8 in each group). (A) Hepatic immunofluorescence of PRR (red), α-SMA (green), and DAPI (diamidino-2-phenylindole, nuclear stain, blue) and (B) α-SMA immunohistochemical staining of liver sections in the 4 groups. ∗P < .05 vs mice treated with vehicle and scramble virus. #P < 0.05 vs mice treated with TAA and scramble virus. Scale bars: 100 μm.