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. 2021 Aug 6;19:335. doi: 10.1186/s12967-021-03003-0

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 7 — BCL7A is an independent predictor of response to TMZ in glioma. A, B Kaplan‒Meier survival plots depicting survival of (IR only)-treated or (TMZ + IR)-treated LGG patients with different BCL7A expression in TCGA-LGG. C, D GSE107850 was used to verify the association between BCL7A and prognosis of LGG patients with different treatments. E, F Effect of BCL7A on prognosis of GBM patients treated with IR or TMZ + IR. G Correlation between BCL7A and MGMT in GBM in four public datasets. H Expression of BCL7A in patients with different MGMT promoter methylation. I, J Effect of BCL7A on prognosis of LGG and GBM patients separated by MGMT promoter methylation status. HR, hazard ratio. IR, ion radiotherapy. *, p < 0.05. ns, no significance