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. 2021 Apr 19;71(8):1636–1647. doi: 10.1007/s12031-021-01832-8

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — Effect of PI3K/AKT activator on chemosensitivity to paclitaxel and stemness in TNC knockdown glioma cells. U251 cell relative cell viability (a), sphere number per 1000 cells (b) and CD44⁺CD133⁺ cell percentage (c, d) among the KD-NC, KD-TNC, KD-NC&740 Y-P and KD-TNC&740 Y-P groups. PI3K phosphatidylinositol 3-kinase; AKT protein kinase B; TNC tenascin C; KD knockdown; NC negative control