Mice from heterozygous intercrosses for the tm1b loss of function allele
or the E373Q catalytic mutant allele, were challenged with 10 weeks of high fat
diet to stimulate atherosclerosis on the ApoE KO background.
The mice were euthanized at 20 weeks of age for evaluation of atherosclerosis. A
and D, Representative photomicrographs of Oil-Red O staining and quantitative
analysis of atherosclerotic lesion area in the aortic arch. B, male WT vs
Adamts7 +/− (HET) vs Adamts7
−/− (KO), n=14 to 18 per group; C, female WT vs HET vs KO, n=10 to
19 per group; E, male WT vs +/EQ vs EQ/EQ, n=9 to 14 per group; F, female WT vs
+/EQ vs EQ/EQ, n=11 to 17 per each group. Data points represent individual
animals, error bars indicate means ±SEM, and two-way ANOVA with
Tukey’s test was applied. Normality was tested by Kolmogorov-Smirnov
test. *P<0.05, **P<0.01.