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. 2021 Jul 26;12:704550. doi: 10.3389/fneur.2021.704550

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Copyright © 2021 He, Chen, Zhong, Yang, Wang, Chen and Guo.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Expression pattern of ATG16L1, LC3, and P62 in TLE patients, the mouse TLE model, and BV2 cells. (A) Temporal lobe tissues obtained from patients with TLE and controls, (B) mouse hippocampal tissues, and (C) BV2 cell lines were analyzed to assess the ATG16L1, LC3, and P62 protein expression levels through western blotting. Statistically significant differences were determined using the unpaired t-test. Data are presented as means ± SD. *p < 0.05; **p < 0.01; ***p < 0.001; ns, not significant (n = 3 per group). miR, microRNA; TLE, temporal lobe epilepsy; SD, standard deviation; KA, kainic acid; ns, not significant; LC3, light chain 3.