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. 2019 Dec 17;1(2):196–205. doi: 10.1016/j.jaccao.2019.09.008

Table 3.

Age at Tumor Diagnosis and PPCM, Baseline LVEF at PPCM Diagnosis, LVEF After Anticancer Treatment, and Genetic Variants in PPCM Patients With Cancer After PPCM

Patient # Tumor Diagnosis Age at Tumor Diagnosis (yrs) Age at PPCM Baseline LVEF LVEF After Cancer Therapy Anticancer Therapy Genetic Variant (P/LP/VUS)
09 Breast cancer 48 36 40 46 Antihormone therapy, surgery, radiation TTN (P)
ATM (P)
ERCC5 (P)
13 Breast cancer 44 40 24 52 Chemotherapy (paclitaxel), antihormone therapy, surgery, radiation TTN (VUS)
POLD1 (VUS)
14 Breast cancer 37 36 20 59 Chemotherapy (paclitaxel, carboplatin), surgery TTN (LP)
NBN (P)
15 Colorectal cancer 33 31 30 60 Surgery Not performed
16 Prolactinoma 30 30 12 HTX No antitumor therapy Not performed
17 Ovarian cancer 36 31 29 55 Surgery RYR1 (VUS)
18 Microprolactinoma 31 31 30 58 No antitumor therapy but bromocriptine Not performed
19 Cervical cancer 45 39 30 50 Surgery Not performed
20 Breast cancer 51 39 22 50 Chemotherapy: tamoxifen; radiation; surgery Not performed
21 Cervical cancer 41 40 32 55 Surgery Not performed

Cardiac function at the time of diagnosis and after cancer therapy in PPCM patients with malignancies after PPCM. PPCM patient 9 had Hodgkin lymphoma before PPCM and breast cancer after PPCM (see Table 2). Variants identified by exome sequencing are presented with their corresponding American College of Medical Genetics and Genomics class. All LP/P variants are in bold.

ATM = TM serine/threonine kinase; ERCC5 = excision repair cross-complementation group 5; HTX = heart transplantation; LP = likely pathogenic; LVEF = left ventricular ejection fraction; NBN = nibrin; P = pathogenic; POLD1 = polymerase delta 1; PPCM = peripartum cardiomyopathy; RYR1 = ryanodine receptor 1; TTN = titin; VUS = variant of unknown significance.