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. 2021 Jul 28;48(3):182. doi: 10.3892/ijmm.2021.5015

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Copyright: © Xie et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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SNHG11 knockdown inhibits PCa cell proliferation, migration and invasion. (A) SNHG11 expression levels were increased in PCa tissues in comparison with those in normal tissues. (B) SNHG11 expression was significantly increased in PCa cell lines (LNCaP, C4-2, PC3, DU145) compared with normal prostate epithelial cell line (RWPE-1). (C) SNHG11 knockdown efficiency in PC3 and DU145 cells detected using RT-qPCR. (D) Colony formation assays suggested that the silencing of SNHG11 suppressed the conony-forming of PC3 and DU145 cells. (E) SNHG11 silencing inhibited the invasion and migration of PC3 and DU145 cells. ^**P<0.01 and ^***P<0.001 in comparison with the control group. SNHG11, lncRNA small nucleolar RNA host gene 11; PCa, prostate cancer.