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. 2021 Jun 3;12(8):1306–1315. doi: 10.1111/jdi.13571

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© 2021 The Authors. Journal of Diabetes Investigation published by Asian Association for the Study of Diabetes (AASD) and John Wiley & Sons Australia, Ltd

This is an open access article under the terms of the http://creativecommons.org/licenses/by-nc-nd/4.0/ License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non‐commercial and no modifications or adaptations are made.

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N6‐methyladenosine (m6A) methylation regulates adipogenesis by various mechanisms. Writers, erasers and readers can modulate m6A modification, subsequently affecting the decay and translation of adipogenesis‐associated regulators, finally regulating adipogenesis. In addition, some regulator and natural compounds, such as nicotinamide adenine dinucleotide phosphate (NADP), zinc finger protein 217 (ZFP217), growth differentiation factor 11 (GDF11) and epigallocatechin gallate (EGCG), can also modulate m6A modification and affect adipogenesis.