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. 2021 Jul 27;11:637247. doi: 10.3389/fonc.2021.637247

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Copyright © 2021 Kim, Kim, Park, Nam, Sung, Bae, Choi, Yoon, Hur and Jang

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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SMAD2 is a direct target of exosomal miR-125b in recipient HCC cells. (A) Two potential seed regions (gray) indicate the binding site of miR-125b in SMAD2 3′ UTR. (B) Western blot assay showing decreased protein expression of SMAD2 after treatment with Exo-125b compared to that after treatment with Exo-NC. (C) Statistical analysis of SMAD2 protein expression in recipient HCC cells. Data are presented as mean ± SEM. *p < 0.05 vs. Exo-NC.