Skip to main content
. Author manuscript; available in PMC: 2022 Aug 1.
Published in final edited form as: Cell Biochem Funct. 2021 Jun 15;39(6):802–812. doi: 10.1002/cbf.3652

Figure 5. Increased tumor expression of high affinity glucose transporter GLUT1 but unaltered MCT1 in cancer cachexia.

Figure 5.

(A) Blots of monocarboxylate and glucose transporters in colon-26 tumor tissue lysates from moderate and severely cachexic mice. (B) Monocarboxylate transporter-1 (MCT1) was detected in colon-26 tumors, but not differentially affected by cachexia. (C) Glucose transporter-1 (GLUT1). (D) Glucose transporter-2 (GLUT2). Data shown as mean±SE. WS=PBS-injected weight-stable mice (n=4); TB-WS=colon-26 (C26) tumor bearing mice that are weight-stable (n=6); Mod=C26 mice with moderate cachexia (10% weight loss) (n=7); Sev=C26 mice with severe cachexia (20% weight loss) (n=6). Data analyzed by one-way ANOVA and Tukey’s multiple comparisons test. P<0.05*.