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. 2021 Mar;21(1):82–95. doi: 10.4314/ahs.v21i1.13

Table 1.

Descriptive characteristics of included articles

References Participants n Age Duration, design Intervention Control Available Outcomes Limitation
Szwejkowski et
al [22], 2013
Patients with
hyperuricemia and
T2DM
66 (59
completers)
64.63±8.79
years
9 months,
RCT
Allopurinol 600
mg twice per
day (n=33)
Placebo (n=33) Difference 600 mg allopurinol versus control
(95% CI):
UA (µmo/l): -251.00 [-313.02, -188.98]
FPG (mmol/l): -0.43 [-2.39, 1.53]
FINS (µU/ml): 4.77 [-4.27, 13.81]
Not adjusted for diabetes duration, kinds
or dosage of hypoglycemic agents. Not
aims to evaluate the effect of uric acid-lowering
therapy on β-cell function or
insulin sensitivity.
Perez-Pozo et
al [23], 2010
Participants were
administered
fructose
200 g daily
83 (74
completers)
40.65 years
(mean 51± 1.3
years)
2 weeks, RCT Allopurinol 200
mg/d (n=38)
No treatment
(n=36)
Difference 100mg allopurinol versus control
(95% CI)
UA (µmo/l): -178.00 [-182.29, -173.71]
FPG (mmol/l) : 0.10 [0.07, 0.13]
(µU/ml): -0.58 [-0.85,-0.31]
HOMA-IR : -0.39 [-0.85, 0.07]
Participates were induced
to hypeluricemia by fructose. Not
adjusted for diabetes duration, kinds or
dosage of hypoglycemic agents. Not
to evaluate the effect of uric acid-lowering
therapy on β-cell function or
insulin sensitivity.
Takir et al [24],
2014
Patients with
hyperuricemia
73 50.76 ± 13.78
years
3 months, RCT Allopurinol 300
mg/d (n=40)
No treatment
(n=33)
Difference 300 mg allopurinol versus control
(95% CI):
UA (µmo/l): -72.00 [-95.86, -48.14]
FPG (mmol/l) :-0.40 [-0.61,-0.19]
FINS (µU/ml): -2.00 [-3.99,-0.01]
HOMA-IR : -3.40 [-5.22, -1.58]
Choice of allopurinol versus control was
performed by the treating physician.
Hence, it was not a pure randomization.
Liu et al [25],
2015
Patients
with T2DM and
hyperuricemia
176 (152
completers)
50.5 ±
10.49 years
3 years, RCT Allopurinol
(starting from
100 mg/day)
adjust to SUA
(n=88)
No
treatment when
the SUA was
less than
476 µmol/L
(n=88)
Difference allopurinol versus
control (95% CI)
UA (µmo/l): -138.00 [-142.13, -133.87]
FPG (mmol/l) : 0.01 [-0.11, 0.13]
FINS (µU/ml): -0.51 [-0.73, -0.29]
HOMA-IR : -0.67 [-0.99, -0.34]
Open-label design and the lack of a
placebo control. Not adjusted
for diabetes duration, kinds or dosage
of hypoglycemic agents. Not aims to
evaluate the effect of uric acid-lowering
therapy on β-cell function or insulin
sensitivity.
Ding et al [26],
2012
Patients with
hyperuricemia
60 48.0±11.7 years 3 ∼ 8 months
(mean 4.3
months), RCT
Allopurinol 100
mg twice a day
(n=30)
No treatment
(n=30)
Difference 200mg allopurinol versus control
(95% CI):
UA (µmo/l): -140.6 [-2.63,-1.69]
FPG (mmol/l) : 0.17 [-0.17,0.51]
FINS (µU/ml): 7.28 [5.62, 8.94]
HOMA-IR: 0.16 [-0.34, 0.67]
Unclear for random sequence
generation or double-blind design. Not
adjusted for kinds or dosage of
hypoglycemic agents.
Le et al [27],
2013
Patients with
hyperuricemia and
IGT
40 44.65±2.27
years
6 months, RCT allopurinol 100
mg three times
a day (n=20)
Low purine and
diabetes diet
(n=20)
Difference 300mg allopurinol versus
control (95% CI)
UA (µmo/l): -203.00 [-221.97, -184.03]
FPG (mmol/l) : -0.66 [-0.86, -0.46]
FINS (µU/ml): -10.56 [-13.39, -7.73]
HOMA-IR : -3.56 4.59, -2.53]
Unclear for random sequence
generation or double-blind design. Not
adjusted for kinds or dosage of
hypoglycemic agents.
Ogino al [28],
2016
Patients with
hyperuricemia
14 60±5 years 8 weeks,
randomized
crossover
study
Benzbromarone
50 mg/d (n=7)
Placebo (n=7) Difference 50 mg benzbromarone versus
control (95% CI):
UA (µmo/l): -128.90 [-156.88, -100.92]
FPG (mmol/l): -0.33 [-0.56, -0.10]
FINS (µU/ml): -7.80 [-9.26, -6.34]
HOMA-IR : -1.22 [-1.72, -0.72]
Participates are patients with CHF and
under the treatment of ACEIs, which
may affect insulin sensitivity. Excluded
diabetes mellitus and antidiabetic
therapy. Short duration and small
sample size. Not aims to evaluate the
effect of uric acid-lowering therapy on
β-cell function or insulin sensitivity.

Abbreviations: T2DM, Type 2 diabetes mellitus; RCT, randomized controlled trial; UA, uric acid; FPG. fasting plasma glucose; FINS, fasting insulin; HOMA-IR, homeostasis model assessment of insulin resistance; IGT, impaired glucose tolerance; CHF, chronic heart failure; ACEIs, angiotensin converting enzyme inhibitors.