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. 2021 Aug 11;11:16293. doi: 10.1038/s41598-021-95771-4

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Viral load of lung tissue of mice infected with H7N9/PR8 recombinant viruses treated with oseltamivir phosphate. The lung tissues of C57 mice infected with 2MLD₅₀ H7N9/PR8 recombinant viruses were titrated 2, 4 and 6 dpi (n = 3/time-point). Mice in the drug treatment group: 20 mg/kg/day oseltamivir phosphate by gavage from 0 to 4dpi; Mice in the non-drug group: PBS by gavage from 0 to 4 dpi. (A–D) Comparison of mice infected with H7N9/PR8 recombinant viruses in the drug treatment group or non-drug group. (E) Comparison of mice infected with rg006NA/PR8 (NA), rg006NA119V/PR8 (119V) and rg006NA222L-119V/PR8 (222L-119 V) virus after oseltamivir administration. *P < 0.05. ^#P < 0.05. ^##P < 0.05. ^###P < 0.05.