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. 2021 Aug 12;22:279. doi: 10.1186/s12882-021-02477-w

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — Typical pathological features of IgG4-Tubulointersitial nephritis and immunohistochemistry staining of surface biomarkers of plasma cells in renal biopsy. The renal interstitium is infiltrated by plasma cells and lymphocytes predominantly with fibrosis on periodic acid-Schiff (PAS) staining. Original magnification 100x (a) and 400× (b). TBM electron-dense deposits were seen under electron microscope. Original magnification 8000x (c). Marked increase in IgG4-positive plasma cells was seen in the infiltrated cells on immunohistochemistry staining. Original magnification 400× (d). CD38-positive plasma cells (e), CD138-positive plasma cells (f), CD56-positive plasma cells (g), and MUM1-positive plasma cells (h) were seen in the interstitium on immunohistochemistry staining. Original magnification 400 ×