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. 2021 Aug 4;2021:5527387. doi: 10.1155/2021/5527387

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Copyright © 2021 Jun Wang et al.

This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Overexpression of PLEK2 promoted gastric cancer proliferation, colony formation, and cell migration. (a) The overexpression of endogenous PLEK2 was confirmed at the mRNA and protein levels. (b) The overexpression of PLEK2 increased the cell proliferation rate in MGC803 and SGC7901 cells. (c) The overexpression of PLEK2 enhanced the colony formation ability of gastric cancer cells. (d) The overexpression of PLEK2 accelerated gastric cancer cell migration. (e) The overexpression of PLEK2 enhanced gastric cancer cell wound healing ability. ^∗P < 0.05, ^∗∗P < 0.01, and ^∗∗∗P < 0.001 (t-test).