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Schematic representation of bimekizumab and sonelokimab’s molecular structures. (a) Bimekizumab has an antigen-binding site that neutralizes interleukin (IL)-17A, IL-17F and their heterodimers (dual specificity). (b) Sonelokimab is a trivalent camelid nanobody comprised of an N-terminal moiety that binds specifically to IL-17F, a central moiety binding to serum albumin for stabilization and a C-terminal moiety binding IL-17A and IL-17F (bispecific inhibitor).
