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. 2021 Aug 17;12(9):799. doi: 10.1038/s41419-021-04081-0

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — A Representative IHC staining for STAT3, LDHA, and FISH staining for LINC00671 in PTC patients. Scale bar, 50 μm. Right panel showing the percentage of specimens with low or high LDHA and STAT3 expressions in the low or high LINC00671 expression groups. CASE 1 and CASE 2 refer to two representative samples categorized by low and high LINC00671 expression. B The correlation of glucose uptake in thyroid cancer patients with different expressions of LDHA, STAT3, and LINC00671 using the Mann–Whitney U test. CASE 1 and CASE 2 refer to two representative samples categorized by low and high FDG uptake. Scale bar, 50 μm. C Graphical abstract underlying the role of the STAT3/LINC00671/LDHA axis in regulating PTC growth and lung metastasis.