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. 2021 Jan 5;45(4):fuaa072. doi: 10.1093/femsre/fuaa072

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© The Author(s) 2021. Published by Oxford University Press on behalf of FEMS.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 2. — Novel therapies interfering with pneumococcal virulence. (A), Drug targets involved in biofilm formation are targeting quorum-sensing mechanisms. (B), Drug targets present on/in individual pneumococci. Drugs specific for these targets aim at inhibition of polysaccharide capsule, pneumolysin and LytA and modification of the pneumococcal cell wall. QS: quorum sensing, LMIP: linear molecularly imprinted polymer, PS: polysaccharide, UDPG:PP: uridine diphosphate glucose pyrophosphorylase, PgdA: peptidoglycan N-acetylglycosamine deacetylase A, AMPs: antimicrobial peptides, PLY: pneumolysin.