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. 2021 Aug 18;16:512. doi: 10.1186/s13018-021-02666-1

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — The ARC from the DDH group was remarkably thickened and had low miR-1-3p expression. A The successful induction of DDH in New Zealand white rabbits, as evidenced by X-ray. B–D The ARC in the DDH model was remarkably thicker than that of the untreated control limb, as evidenced by gross specimen observation, MRI imaging, and HE staining. Scale bar: 100 μm. F QPCR confirmed the reduced miR-1-3p expression in the DDH ARC samples versus the controls. (Data represents mean ± SD, n = 3. *P < 0.05.)