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. 2021 Aug 19;4:933. doi: 10.1038/s42003-021-02448-9

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© The Author(s) 2021, last corrected publication 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — a HU-induced the elevation in brain Ca²⁺ levels. (n = 6). HU hindlimb unloading. b, c Changes in the contents and activities of SSAO in the cerebellum of HU mice. (n = 6). SSAO semicarbazide-sensitive amine oxidase. d, e Changes in the contents and activities of brain FDH. (n = 6). FDH formaldehyde dehydrogenase. f Blood SSAO activity detected using a mouse SSAO kit. (n = 6). g SSAO (green) located in the cerebellum. DAPI (blue, a dye for nuclear). h Statistical analysis of cerebellar SSAO expression (n = 8). i Model of stress-induced FA generation. FA formaldehyde, FDH formaldehyde dehydrogenase, SSAO semicarbazide-sensitive amine oxidase. Error bars show the mean ± SEM; *p < 0.05, ***p < 0.001.