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. 2021 Aug 19;23:98. doi: 10.1186/s12968-021-00789-2

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 4 — Qualitative intracardiac flow profile via velocity vector fields overlaid onto cine imaging. Blue arrows are pointed to larger vorticity magnitude, red arrows pointed to smaller vorticity magnitude. In rTOF patients, there is a dominant vortex generated from pulmonary insufficiency, best visualized in short axis (SAx) and modified right ventricular outflow tract (RVOT) view. This vortex is separate from the smaller vortices generated by the tricuspid inflow, best visualized in the 4-chamber (4Ch) view. For right ventricular dilation (RVD patients), a larger vortex is generated from tricuspid inflow and propagates into the RVOT, but the overall flow topology is unchanged when compared to controls